Welcome to the Friedrich Miescher Laboratory of the Max Planck Society
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Research overview
Find out more about our scientific research areas.
Scientific Facilities and Services
The FML research groups have a wide range of specialized high-end instrumentation in our dedicated research facilities.
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Plan your career at the Institute.Research Groups
Structural Regeneration
Dr. Can Aztekin
Max Planck Research Group Leader
Why do only a few species regenerate their limbs? How do they restore a lost limb? What’s different in mammals? To find answers, we compare cell types between regeneration-competent tadpoles and regeneration-incompetent mice.
Synthetic Biology Approaches to Pattern Formation
Dr. Beatrice Ramm
Max Planck Research Group Leader
Our group contributes to the understanding of pattern formation in biology with an interdisciplinary approach that sits at the interface of biochemistry, biophysics and synthetic (developmental) biology.
Evolutionary Genomics of Complex Traits
Dr. Luisa Pallares
Max Planck Research Group Leader
We study the genetic basis of phenotypic
variation between individuals, and the role that those genetic effects play in the robustness and evolvability of natural populations of the fruit fly, Drosophila melanogaster.
Structural Biochemistry of Meiosis
Dr. John Weir
Max Planck Research Group Leader
Meiosis is the process by which the chromosomes from each parent are properly segregated into gametes (e.g. eggs and sperm). Our group examines the mechanisms that control this critical process in the development of new life.
News
Silvia Da Pra, postdoctoral researcher at the Friedrich Miescher Laboratory of the Max Planck Society, has been awarded a 2026 EMBO Postdoctoral Fellowship to study hormonal control of limb regeneration.
This year’s Schering Young Investigator Award has been awarded to Can Aztekin, a Max-Planck-Research Group Leader at the Friedrich Miescher Laboratory of the Max Planck Society in Tübingen, to recognize his significant work in the field of ...
Researchers show that mammals briefly activate regenerative programs after injury but shut them down too fast. Altering oxygen sensing keeps cells in a repair-ready state.
